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Comparison with an MTP inhibitor.We investigated

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the effects

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of Atorvastatin ( Lipitor ), a widely used 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) suicide prescription drugs ( Lipitor ) increases hepatic fatty acid beta-oxidation

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in sucrose-fed rats.

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Cholesterol concentration was determined in 7 LDL subfractions isolated by density gradient ultracentrifugation before and during Atorvastatin ( Lipitor )

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treatment.

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Effect of Atorvastatin ( Lipitor ) on low-density lipoprotein subtypes in patients with different forms of hyperlipoproteinemia and control subjects.Atorvastatin

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( Lipitor ) is a potent hydroxy-methyl-glutaryl-coenzyme

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A (HMG-CoA) reductase inhibitor that decreases low-density lipoprotein (LDL) cholesterol and triglyceride concentrations, but little is known about its effects on LDL subtype distribution

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in different types of hyperlipoproteinemia.

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Thus, we evaluated the influence of Atorvastatin ( Lipitor ) (10 mg/d, 4 weeks) on lipid concentrations and LDL subtype distribution in patients with hypercholesterolemia (n 9; LDL cholesterol, 227 /- 30 mg/dL; triglycerides, 137 /- 56 mg/dL), patients with type 2 diabetes and dyslipoproteinemia (n 11; LDL cholesterol, 163 /- 34 mg/dL; triglycerides, 260 /-

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147 mg/dL), and controls (n 10; LDL cholesterol, 116 /- 20 mg/dL; triglycerides, 130 /- 47 mg/dL). These results indicate that the activation of hepatic beta-oxidation by Atorvastatin ( Lipitor ) may contribute to the decrease in hepatic triglyceride concentration, leading to its hypotriglyceridemic effect. In the Atorvastatin ( Lipitor )-treated groups, a significant

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relationship was seen not only between hepatic beta-oxidation activity and hepatic triglyceride concentration (R(2) 0.535, P < 0.01), but also between hepatic and plasma triglyceride concentrations (R(2) 0.586, P < 0.01). Atorvastatin ( Lipitor ) (30 mg/kg) increased hepatic beta-oxidation activity by 54% (P < 0.01), while BMS-201038 (0.3 mg/kg) had no significant effect. The decrease in plasma triglyceride concentration and post-Triton very low-density lipoprotein (VLDL) triglyceride

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concentration, a measure of hepatic triglyceride secretion, by Atorvastatin ( Lipitor ) (30 mg/kg p.o.) for 2 weeks was to approximately the same degree as those by BMS-201038 (0.3 mg/kg). No effect of Atorvastatin ( Lipitor ) on hepatic fatty acid synthesis was observed.

Atorvastatin ( Lipitor ) decreased LDL cholesterol (-36%, -28%, and -41%, all P.